Selank
A synthetic tuftsin analog that modulates GABAergic signaling and inhibits enkephalin-degrading enzymes to produce sustained anxiolytic, nootropic, and neuroinflammation-reducing effects without tolerance or dependence.
A synthetic tuftsin analog that modulates GABAergic signaling and inhibits enkephalin-degrading enzymes to produce sustained anxiolytic, nootropic, and neuroinflammation-reducing effects without tolerance or dependence.
Selank inhibits enkephalin-degrading enzymes, prolonging endogenous opioid peptide activity and supporting mood regulation. It potentiates GABAergic transmission without inducing receptor downregulation or dependence, and normalizes IL-6 and interferon-γ levels to calm neuroinflammation. Enhanced BDNF expression in limbic regions supports long-term cognitive clarity and emotional balance.
- [1]Semenova TP, et al. (2010). Selank Modulates the Expression of Genes Involved in GABAergic and Serotonergic Neurotransmission. Bulletin of Experimental Biology and Medicine, 150(4):484-487.
- [2]Zozulya AA, et al. (2006). The comparison of noopept and piracetam in the treatment of intellectual-mnemic disturbances in patients with organic emotionally labile (asthenic) disorders. Bulletin of Experimental Biology and Medicine, 141(3):273-276.
- [3]Kozlovskaya MM, et al. (2014). Selank and short peptides of the tuftsin family in the regulation of adaptive behavior in stress. Pharmaceutical Chemistry Journal, 48(9):591-598.
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